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Additionally, insulin from MIF-deficient mice experienced a different conformation or posttranslational modification as compared to insulin from wildtype mice. MIF basically stimulated insulin hexamer formation in cell-free systems [100]. So, plainly MIF can work as an adaptor protein for insulin oligomerization. Insulin was also discovered to co-i
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A new discovery highlighted a novel part for intracellular MIF in regulating the accumulation of misfolded Cu/Zn superoxide dismutase (SOD1). Mutations in SOD1 are connected with 20% in the conditions of familial amyotrophic lateral sclerosis, which is characterized by loss of motor neurons [seventy two]. b A summary with the normalized MIF, D-DT
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